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Date & Time: September 6, 4pm
Location: Faculteit Sociale Wetenschappen, Pieter de la Court gebouw, Wassenaarseweg 52, room SB-45 

 

 

 

Date and time: January 23, 2018, 11am

Program:

11:00 - 11:45 
The neural underpinnings of empathy - from affect sharing to self-other distinction, Prof. Dr. Claus Lamm 
In my talk, I will highlight how recent research in the field of social neuroscience advances our understanding of the multi-faceted construct of empathy. I will in particular address two key components, affect sharing, and self-other distinction, and how they are affected by healthy aging as well as acute psychosocial stress.

11:45 - 12:30 
Neuroimaging research in conduct disorder: Preliminary findings from the FemNAT-CD European Consortium, Dr. Stephane De Brito 
Antisocial behaviour is a major public health concern. Children and adolescents with conduct disorder have a poor prognosis with negative adult outcomes that frequently include criminality, alcohol and substance abuse, and poor mental and physical health. In this talk, I will present preliminary structural and functional neuroimaging findings from the FemNAT-CD study, a large multisite European consortium focusing on the environmental and neurobiological factors underpinning sex differences in conduct disorder.

12.30 - 13.15 | Lunch

13.15 - 14.00
When neuroscience met psychopathy: a tale of frenemies, Dr. Inti Brazil 
The incorporation of methods borrowed from experimental psychology and neuroscience to study psychopathy has led to major advances. However, the current rate at which neuroscience develops is much faster than the developments in psychopathy research. In this talk, I will discuss how the fast development of neuroscience has led to major breakthroughs in psychopathy research, but is also creating new problems for the field.

Location: Klein Auditorium, Academiegebouw Universiteit Leiden, Rapenburg 73, Leiden

Register here

Bridging scales with neuroimaging: challenges and opportunities

Date and time: 30 november 2017, 4:30pm

Neuroimaging provides unique opportunities to address one of the grand challenges in neuroscience: relating structure and function over many orders of magnitude. I will focus on different aspects of scale (space, time and people), and how the next generation of MRI methods will enable us to face this challenge. Themes will include adopting a synergistic approach to acquisition and analysis; relating neuroimaging tools to complementary techniques; and the new era of population neuroimaging.

Karla Miller develops novel methods for acquiring, reconstructing and analyzing MRI scans of the brain. Her early research focused on techniques for studying brain function and connectivity. More recently, she has also begun to develop techniques for studying tissue microstructure with MRI, including the use of complementary technologies like microscopy.

Location: Faculty of Social and Behavioural Sciences, Pieter de la Court gebouw, Wassenaarseweg 52, room SA-41

Integrating measures of genomic risk in studies of child neurodevelopment: precision medicine in child psychiatry?

Date and time: 19 October 2017, 1.00pm

How does our early experience influence vulnerability for later adverse mental health outcomes?  This is the question that drives Dr. O’Donnell’s multidisciplinary research program at McGill University. He combines psychiatric, genetic, epigenetic and epidemiological methods to better understand the biological embedding of early adversity and its association with altered child neurodevelopment.  Specific areas of interest include maternal perinatal mental health and child neurodevelopment, as well as epigenetic analyses of early intervention programs, which seek to buffer the effects adversity experienced early in life. The goal is integrate relevant biological measures into studies of the child neurodevelopment to better identify and treat at-risk individuals, and thus help each child reach their full developmental potential. The current lecture will include a discussion about the reality of precision medicine in child mental health with some examples from his own studies.

Location: Faculty of Social and Behavioural Sciences, Pieter de la Court gebouw, Wassenaarseweg 52, room SA-41

Date & Time: 29 June 2017, 4.30 pm
Location: FSW, Pieter de la Court gebouw, Wassenaarseweg 52, College room SA41


The LIBC annually organises a LIBC member day. During this meeting researchers share their research findings with colleagues from different departments via lectures and poster presentations.

 

Date: to be announced
Time: to be announced
Location: to be announced

 

 

Davood  Ghara Gozli (Cognitive Psychology) has received a Veni grant. As such, he is officially recognised as a promising young scientist by science funding agency NWO and will receive financial support to further develop his ideas. read more

Judging, Feeling, Thinking: What makes us special?

Date & Time: Thursday November 5, 2015, 4.00 pm
Location: University Leiden, Lipsiusgebouw, Cleveringaplaats 1, room 011

Human brains closely resemble those of other primates, yet we do many nifty things that they do not. It’s often said that we humans are distinguished by our “humanity”, by our moral sensibilities. But research on moral cognition says that human morality is not the product of a distinctive moral faculty. Instead, moral judgment and behavior depend on the coordinated interaction of multiple neural systems, none of which appears to be specifically dedicated to moral cognition and all (or nearly all?) of which appear to be shared with other animals. What, then, makes us special? I’ll provide a selective overview of research on moral cognition, with an emphasis on the roles played by domain-general cognitive systems. I’ll then discuss some new research investigating what I take to be an essential ingredient—if not the essential ingredient—for uniquely human intelligence: the ability to rapidly and flexibly combine multi-modal concepts to form complex thoughts. Please check out his website for more info: http://www.joshua-greene.net/research/moral-cognition/

In 47,XXY (also referred to as Klinefelter syndrome), boys are born with an extra X chromosome. The physical, cognitive and behavioral consequences of this condition vary, but 47,XXY is associated with an increased risk of psychopathology. The mechanisms underlying the impairments in 47,XXY have not yet been clearly defined. Knowledge of neural mechanisms that may lead to these problems, could benefit the development and selection of interventions. For this reason, the first aim of this dissertation was structural and functional brain mapping in a sample of boys with 47,XXY.

5 – 25% of individuals with 47,XXY are diagnosed with autism spectrum disorders (ASD). However, even without a formal ASD diagnosis, prominent social/communicative difficulties may be present. In spite of this overlap in autism symptomatology, distinct differences in behavioral features and underlying cognitive mechanisms have also been reported. This suggests the existence of various pathways to social dysfunction, and possibly differential susceptibility to treatment. As this may have important implications for mental health care strategies, the second aim of this study was to compare brain structure and function of boys with 47,XXY, to that of boys with idiopathic ASD.

Date: 27 november 2014, 12.30 hrs
Location: Onderwijsgebouw, V3-36

Typically, adolescence is associated with an increase in risk-taking behavior. Adolescent brain development is often described in terms of faster developing reward regions versus protracted development of the prefrontal regions (Somerville et al, 2010). This developmental mismatch results in heightened sensitivity for rewards during adolescence. Indeed studies confirm heightened sensitivity of the reward system (Braams et al, 2014; Galvan et al, 2006). However, the adolescent reward system is not always hyperactive, but depends on social context. Receiving rewards for liked and disliked others results in differential striatum responses, with higher responses when winning for a liked versus a disliked other (Braams, 2014).
It is currently unclear which factors influence the striatum response to rewards for self and others. Pubertal development, including hormonal measures such as testosterone levels, has been proposed as one potential underlying biological mechanism driving these different responses. A second important focus point is individual difference measures such as reward sensitivity and risk-taking behavior in the lab as explaining the striatum responses across studies. To understand striatum responses during development a large sample with multiple measures is necessary.
In this study we measured neural activation during a reward processing task combined with pubertal measures and individual difference measures. Data were collected twice with a two year interval in between sessions. On the first time point data were collected from 299 participants aged 8-26 and from 286 participants aged 10-28 on the second time point. In the reward task, participants not only played for themselves, but also for their best friend. This study design with a large, longitudinal sample, allows us to test which factors are related to adolescent reward processing across different social contexts and across time. These results provide important insights in development of factors related to adolescent risk-taking behavior.

Date: 20 november 2014, 12.30 hrs
Location: Onderwijsgebouw, V4-18/22

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